Tuesday, July 12, 2011

CASE: Old age Huge Recurrent strangulated incisional hernia + HTN + IHD + Obesity^-www.drkeyurbhatt.in*

72 yrs female with Huge Recurrent strangulated incisional hernia + HTN + IHD + Obesity
came with acute onset vomiting , fever, distension, constipation, tachycardia
Past history: Lap chole and paraumbilical hernia repair 1.5 yrs back. (mesh plasty, lap)

CECT:



SURGERY:

anterior abdominal wall had total 11 defects all transmiting bowel



Discharged after 7 day.

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CASE: Mirizzi syndrome type 1/2 with gangrenous cholecystitis (retro virus positive)^-www.drkeyurbhatt.in*

42 yr male and RV positive for 2 yrs on HAART
Developed sever pain in RHC and diagnosed as acute gangrenous cholecystitis

MRCP :   Phrygian Cap with Mirizzi type 2.





SURGERY: Open cholecystectomy and choledochoplasty:




pt discharged afte 6 days of surgery

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CASE: MINIMAL INVASIVE PANCREATIC NECROSAECTOMY^-www.drkeyurbhatt.in*

28 yrs male with severe nerotizing pancreatitis
with sepsis.  on 28 days of illness


CECT:

SURGERY:
 Lap assisted minimal invasive pancreatic necrosectomy:



pt discharged on POD 7 with drain in situ..

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CASE: LAPAROSCOPIC TRANS PERITONEAL NECROSECTOMY^-www.drkeyurbhatt.in*

Middle aged male with acute necrotizing pancreatitis  (Alcoholic). with persistent fever and nausea.
26 kg of wt loss. and on and off abdominal pain. 2 months following acute attack of necrotizing pancreatitis.

CECT:




SURGERY: TOTAL Laparoscopic Trans peritoneal necrosectomy, lavage and drainage.

opening of lesser sac

stomach lifted and lesser sac entered at the area of necrosis

removal of necrosis
placement of drain

post op
dirty necrotic material

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Tuesday, July 5, 2011

CASE : SPLENIC ARTERY PSEUDOANURISM BLEEDING Status TWICE COILING Status Chronic pancreatitis (alcoholic)^-www.drkeyurbhatt.in*

after 9 months of follow up Pt is doing well and gained 8 kg wt....no further complaints...


previous angio coiling, patent pancreatica
megna branch and partially occluded splenic artery


ligation of splenic artery at its origin before
arteria pancreatic magna branch


pseudoanurism in arteria pancreatica
magna teritory





Pancreatic Pseudoaneurysm: Treatment

  • Transarterial catheter angioembolization with or without endoscopic stent placement. 
o    Endovascular coil embolization has been used extensively in the treatment of visceral artery pseudoaneurysms.
o    Angioembolization is considered much less invasive than surgery. The procedure can be completed quickly and is comfortable for the patient. It also allows the performance of surgery under optimal conditions.
o    The interventional approach has a reported success rate of 67-100% over the past few years.6
o    Most authorities agree that embolization is appropriate when bleeding is diffuse or emanating from the pancreatic head, for unsuccessful surgery, or during postoperative bleeding.
o    Failure results from an inability to selectively catheterize the bleeding vessel or the misplacement or poor placement of embolization material.
o    In addition to rebleeding, complications of this procedure include rupture of the pseudoaneurysm during embolization, arterial perforation by the catheter, intestinal necrosis, and aortic thrombosis.


SURGICAL TREATMENT

The indications for emergent exploratory laparotomy include hemodynamic instability and failure of endovascular techniques to control active hemorrhage.

Basic surgical techniques for controlling hemorrhage from a pancreatic pseudoaneurysm include arterial ligation on both sides of the bleeding sites, pancreatic resection, and intracystic/extracystic multiple ligatures

Clinical features and management of splenic artery
pseudoaneurysm: Case series and cumulative
review of literature

(J Vasc Surg 2003;38:969-74.)

Deron J. Tessier, MD,a William M. Stone, MD,a Richard J. Fowl, MD,a Maher A. Abbas, MD,a James C.
Andrews, MD,b Thomas C. Bower, MD,c and Peter Gloviczki, MD,c Scottsdale, Ariz; and Rochester, Minn

Introduction: Splenic artery pseudoaneurysm is uncommon. We report our institution’s recent 18-year experience with
these aneurysms and review the literature.
Methods: We reviewed the records for 37 patients with visceral artery pseudoaneurysm evaluated at our institution from
1980 to 1998. From this group we found only 10 patients (27%) with splenic artery pseudoaneurysm. We also reviewed
147 cases of splenic artery pseudoaneurysm reported in the English literature.
Results: In this series of 10 patients, 5 were men. Mean age was 51.2 years (range, 35-78 years). Causes of aneurysm
included chronic pancreatitis in 4 patients, trauma in 2 patients, iatrogenic cause in 1 patient, and unknown cause in 3.
The most common symptom was bleeding in 7 patients and abdominal or flank pain in 5 patients; 2 patients had no
symptoms. Aneurysm diameter was known for four pseudoaneurysms, and ranged from 0.3 to 3 cm (mean, 1.7 cm).
Splenectomy and distal pancreatectomy were performed in 4 patients, splenectomy alone in 2 patients, endovascular
transcatheter embolization in 2 patients, and simple ligation in 1 patient. One patient with a ruptured pseudoaneurysm
died before any intervention could be performed; there were no postoperative deaths. Follow-up data were available for 7 patients, with a mean of 46.3 months (range, 4.5-120 months).

Conclusions: Splenic artery pseudoaneurysm is rare and usually is a complication of pancreatitis or trauma. Average aneurysm diameter in our series of 10 patients was smaller than previously reported (1.7 cm vs 5.0 cm). Although conservative management has produced excellent results in some reports, from our experience and the literature, we recommend repair of all splenic artery pseudoaneurysms. 











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